New Research: Aerobic Exercise May Make You A Fat STORING Machine
January 21, 2010 by admin
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New research suggests that doing exercise in your “fat burning zone” will actually reduce the amount of fat that your body burns for the rest of that day. A new study in the Journal of Applied Physiology had participants do 1 hour of cycling at 55% of their aerobic capacity only to find that “24 h fat balance was significantly more positive on [the exercise day] compared to [the non-exercise day].”
Local fitness expert Josef Brandenburg says, “this is what I’ve been telling my clients for years – aerobic exercise makes you fatter.” Brandenburg hypothesizes that the body responds to exercise based on the type of fuel it uses.
In high intensity exercise like sprinting and weight lifting the primary fuel is carbohydrate. Your body responds to carb-dependent exercise by making it easier for your body to replace those carbs (increasing your insulin sensitivity), and increasing your capacity to store carbohydrates (called glycogen).
With low-intensity, aerobic exercise – like that done in the study and during most people’s workouts – the primary fuel is fat. Brandenburg thinks that burning fat as the primary fuel during exercise is the signal to your body to become better at storing fat, hence the results of the study.
He says your body responds by doing the opposite. At the end of a resistance workout you are smaller and weaker than when you started it, but your body responds by doing the opposite – building you up in preparation for the next time it is stressed.
“Research in the International Journal of Sports Nutrition shows that resistance training shifts your body to burn more fat for the 24 hours after your workout unlike with aerobics. I think that since body-fat is fundamentally regulated by the hormone insulin, the intense, carb-dependent exercise like resistance training makes you burn fat all day long because it reduces your insulin levels and improves your carbohydrate metabolism,” says Brandenburg.
To check out the study for yourself, click here.
For free info about what to do with this click here
Josef Brandenburg is 2010, Washington, DC Personal Trainer of the Year Nominee for both Personal Fitness Professional Magazine and The Washington, DC Fitness Association, and author of “The Body You Want”. He shows normal people with hectic lives and average genetics how to create the bodies they want in the time they actually have. To find out more about the 7-Day Free Trial click here. You can also pick up a FREE copy of his brand new CD – “Why Eat Less and Exercise More is The Worst Advice Ever” here..
Can Laughing Help You Lose Weight?
January 15, 2010 by admin
Filed under Uncategorized
I recently ran across a very neat research article in Diabetes Care on the power of laughter.
Japanese researchers took a group of type-2 diabetics and healthy subjects for two days:
Day 1 – they ate a 500 calorie lunch and attended a “monotonous lecture (40 min) without humorous content.”
Day 2 – they ate an identical 500 calorie lunch and attended a 40min comedy show where “the subjects laughed.”
(Funny excerpt: “The subjects estimated their laughter level on a scale of 0–5, and most of them considered that they laughed well (level 4 or 5).” Leave it to a scientist to take the fun out of a comedy show!)
Researchers monitored the subjects bloo d glucose levels for two hours after the and in the people who attended the comedy show glucose levels where significantly lower .
“Negative emotions such as anxiety, fear, and sorrow are known to be factors that elevate the blood glucose level.”
Why Does This Matter To You?
Elevated blood sugar (glucose) levels require that your body secretes insulin to bring them back down. The more insulin you secrete the more your fat cells grow. Anything you can do to reduce your insulin levels (high intensity exercise, low-carb eating) will help to make you leaner.
Apparently, its not just what you eat, but how you eat. If you eat in a stressed out state, that can make your healthy lunch less healthy.
So, lighten up! Pass gas and blame it on somebody else at lunch. Make sure that you are having fun every day – even at work.
You can read the whole article in Diabetes Care right here for free:
http://care.diabetesjournals.org/content/26/5/1651.full
Josef Brandenburg is 2010, Washington, DC Personal Trainer of the Year Nominee for both Personal Fitness Professional Magazine and The Washington, DC Fitness Association, and author of “The Body You Want”. He shows normal people with hectic lives and average genetics how to create the bodies they want in the time they actually have. To find out more about the 7-Day Free Trial click here. You can also pick up a FREE copy of his brand new CD – “Why Eat Less and Exercise More is The Worst Advice Ever” here.
What If Low Carb Is Wrong? Does ASP Prove That Insulin Doesn’t Matter and That Its Calories That Really Count?
June 30, 2009 by admin
Filed under Uncategorized
So there I was on the forums of t-nation.com answering questions about my recent interview piece with Gary Taubes when I started to wonder if this low-carb thing was legitimate after all. “Have I been giving bad advice the whole time? Should I just be telling people to ‘eat less and exercise more?’ But that’s never worked… What I do now works like clock work… But all these posts keep telling me how ‘retarded’ me and Gary are…’”, I worried to myself.

What’s not authoritative and intimidating about being insulted over and over again by anonymous people on a forum? If they can type it on a forum it must be well researched and true! Right?
I did come to my thin-skinned senses eventually, BUT the folks on the t-nation forums did leave me with one question that I wasn’t entirely sure about: Is ASP the overlooked flaw in low-carb dieting?
There were a lot of posts saying in effect: “What Tuabes and the rest of the low-carb community don’t like talking about is a little compound called acylation stimulating protein (ASP) which stores dietary fat in the fat cell with ZERO rise in insulin levels.”
I started doing some digging and found statements like these in peer reviewed research (I’m paraphrasing to make them readable and concise):
#1. “ASP is far more powerful than insulin in stimulating the creation of new body-fat.” (1)
#2. “ASP is released in response to an oral fat load.” (2)
Did I Get Served On A Forum?

I started really getting into ASP research at about 10pm on a Saturday night, and by the time I found the above statements in peer reviewed research I started sweating and my stomach was twisted up in knots. I tried to let it go and hang out with my fiancé, but I just had to know – “What’s the deal with ASP!? Is this something the debunks low-carb?”
So I spent the next 6 hours combing everything that I could get my hands on about ASP, and every time a paper made on of the above statements (#1 & 2 above) I noted the paper they cited. (By the way, my biochemistry textbooks were no help at all.) Pretty soon it became clear that those two statements were pretty much built off of two papers published in 1989:
• Statement #1 above was pretty much built off of a paper titled “Purification and characterization of acylation stimulating protein”, but let’s call it “ASP trumps insulin” for this article.
• Statement #2 above was built off of a paper titled “Metabolic response of acylation stimulating protein to an oral fat load,” but let’s call it “ASP is released in response to fat, not carbs.”
Does ASP Trump Insulin?
For the “ASP trumps insulin paper” the researchers grew fat cells in cultures (outside the body). It is true the addition of ASP REALLY accelerated the creation of new fat (triglyceride) – it was faster than insulin.
HOWEVER, both the ASP and the no-ASP cultures had insulin and carbs added to them. So the water is very muddy:
Could the ASP be a way in which insulin accelerates its activity? Maybe the super deadly combo a high-fat and high sugar meal? Maybe the only thing to learn from this study is that frosting is more fattening than candy? Why not test ASP without insulin and carbs if your goal is to see what the difference between them is?
Or, for that matter, why not just test ASP in actual people instead of cultures? (1)
Is ASP Released In Response to Fat, Not Carbs?
For the “ASP is released in response to fat, not carbs.” The “lipid meal” or “oral fat load” that was used to induce the ASP response was cream + one tbsp table sugar (sucrose) + one tbsp nonfat dry milk.
That mixture is 25% carbs by weight! So, that really doesn’t tell us anything at all.
Why not just have people drink olive oil or melted butter instead of cream plus a bunch of sugary stuff?
Take It Home Gary
I shared my research with Gary Taubes and he had this to say:

Gary and his cat
“Nice to know you’re suitably obsessed Josef.
“One thing to keep in mind in all this is the need to explain the observations, not just work with possible mechanisms that can’t explain anything. So one of the observations is weight loss on a high fat diet — the Atkins diet.
“So if ASP was good at sequestering dietary fat away in the fat tissue without carbs being needed, why would people lose weight when they ate an Atkins diet?
“Another observation we’re trying to understand, as I point out in lectures, is the obesity in poor populations eating low-fat, high carb diets. So there ASP would be irrelevant. Now if we had obesity in populations eating low-carb, high-fat diets, that would be telling and a reason to invoke ASP, but, as far as I know, no such populations exist.
“So maybe ASP plays a role in obesity in rats that are fed high fat diets, but we’re not all that interested in rats.”
Epilogue – Falling Asleep At A Wedding
My obsessive researching kept me up ‘till 4am, but I had to be up at 8am to workout, shower and be at a friend’s wedding the next day. I couldn’t put off my workout and sleep in because I had deadlines to meet that night. So, while everyone else was watching the cake cutting and dancing, I passed out, face down on the table. Even though I woke with drool on my cheek I felt a lot better.

References
#1. J Biol Chem vol. 264, Jan 5, 1989, p.426-430
#2. J lipid res vol 30, 1989 p. 1727-1733
















